Allostasis and allostatic load/Bibliography: Difference between revisions

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*McEwen BS, Lasley EN. (2002) The end of stress as we know it. Washington, D.C: Joseph Henry Press. ISBN 0309076404.
*McEwen BS, Lasley EN. (2002) The end of stress as we know it. Washington, D.C: Joseph Henry Press. ISBN 0309076404.
*Schulkin J. (2003) Rethinking homeostasis: allostatic regulation in physiology and pathophysiology. Cambridge, Mass: MIT Press. ISBN 0262194805.
*Schulkin J. (2003) Rethinking homeostasis: allostatic regulation in physiology and pathophysiology. Cambridge, Mass: MIT Press. ISBN 0262194805.
:*[https://cognet.mit.edu/library/books/view?isbn=0262194805 MIT CogNet: The Brain Sciences Connection]
:*'''<u>From the book announcement website:</u>''' Homeostasis, a key concept in biology, refers to the tendency toward stability in the various bodily states that make up the internal environment. Examples include temperature regulation and oxygen consumption. The body's needs, however, do not remain constant. When an organism is under stress, the central nervous system works with the endocrine system to use resources to maintain the overall viability of the organism. The process accelerates the various systems' defenses of bodily viability, but can violate short-term homeostasis. This allostatic regulation highlights our ability to anticipate, adapt to, and cope with impending future events....In Rethinking Homeostasis, Jay Schulkin defines and explores many aspects of allostasis, including the wear and tear on tissues and accelerated pathophysiology caused by allostatic overload. Focusing on the concept of motivation and its relationship to the central nervous system function and specific hormonal systems, he applies a neuroendocrine perspective to central motive states such as cravings for water, sodium, food, sex, and drugs. He examines in detail the bodily consequences of the behavioral and neuroendocrine regulation of fear and adversity, the endocrine regulation of normal and preterm birth, and the effects of drug addiction on the body. Schulkin's presentation of allostasis lays the foundation for further study.
:*Table of Contents: Preface; Introduction; Allostasis: The Emergence of a Concept; Central Motive States: Feedforward Neuroendocrine Systems in the Brain; Anticipation, Angst, Allostatic Regulation: Adrenal Steroid Regulation of Corticotropin-Releasing Hormone; Normal and Pathological Facilitation of Parturition by a Feedforward Endocrine Mechanism; Addiction to Drugs: Allostatic Regulation under Duress; Conclusion; References; Name Index; Subject Index.
*International Society of Psychoneuroendocrinology, Congress, (34th et al.) (2004) Biobehavioral stress response: protective and damaging effects. New York, N.Y: New York Academy of Sciences.
*International Society of Psychoneuroendocrinology, Congress, (34th et al.) (2004) Biobehavioral stress response: protective and damaging effects. New York, N.Y: New York Academy of Sciences.
*McEwen BS. (2004) The end of stress as we know it. Washington, D.C: Joseph Henry Press.
*McEwen BS. (2004) The end of stress as we know it. Washington, D.C: Joseph Henry Press.

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A list of key readings about Allostasis and allostatic load.
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Books

  • McEwen BS, Lasley EN. (2002) The end of stress as we know it. Washington, D.C: Joseph Henry Press. ISBN 0309076404.
  • Schulkin J. (2003) Rethinking homeostasis: allostatic regulation in physiology and pathophysiology. Cambridge, Mass: MIT Press. ISBN 0262194805.
  • MIT CogNet: The Brain Sciences Connection
  • From the book announcement website: Homeostasis, a key concept in biology, refers to the tendency toward stability in the various bodily states that make up the internal environment. Examples include temperature regulation and oxygen consumption. The body's needs, however, do not remain constant. When an organism is under stress, the central nervous system works with the endocrine system to use resources to maintain the overall viability of the organism. The process accelerates the various systems' defenses of bodily viability, but can violate short-term homeostasis. This allostatic regulation highlights our ability to anticipate, adapt to, and cope with impending future events....In Rethinking Homeostasis, Jay Schulkin defines and explores many aspects of allostasis, including the wear and tear on tissues and accelerated pathophysiology caused by allostatic overload. Focusing on the concept of motivation and its relationship to the central nervous system function and specific hormonal systems, he applies a neuroendocrine perspective to central motive states such as cravings for water, sodium, food, sex, and drugs. He examines in detail the bodily consequences of the behavioral and neuroendocrine regulation of fear and adversity, the endocrine regulation of normal and preterm birth, and the effects of drug addiction on the body. Schulkin's presentation of allostasis lays the foundation for further study.
  • Table of Contents: Preface; Introduction; Allostasis: The Emergence of a Concept; Central Motive States: Feedforward Neuroendocrine Systems in the Brain; Anticipation, Angst, Allostatic Regulation: Adrenal Steroid Regulation of Corticotropin-Releasing Hormone; Normal and Pathological Facilitation of Parturition by a Feedforward Endocrine Mechanism; Addiction to Drugs: Allostatic Regulation under Duress; Conclusion; References; Name Index; Subject Index.
  • International Society of Psychoneuroendocrinology, Congress, (34th et al.) (2004) Biobehavioral stress response: protective and damaging effects. New York, N.Y: New York Academy of Sciences.
  • McEwen BS. (2004) The end of stress as we know it. Washington, D.C: Joseph Henry Press.
  • Schnurr PP, Green BL. (2004) Trauma and health: physical health consequences of exposure to extreme stress. Washington, DC: American Psychological Association.
  • Schulkin J. (2004) Allostasis, homeostasis and the costs of physiological adaptation. Cambridge UK: Cambridge University Press.
  • Eaton WW. (2006) Medical and psychiatric comorbidity over the course of life. Washington, DC: American Psychiatric Pub.