Bariatric surgery/Related Articles: Difference between revisions
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{{r|Glucostatic theory of appetite control}} | {{r|Glucostatic theory of appetite control}} | ||
{{r|Melanocortins and appetite}} | {{r|Melanocortins and appetite}} | ||
{{r|Stress and appetite}} | {{r|Stress and appetite}} | ||
{{r|adipocyte}} | |||
{{r|leptin}} | |||
{{r|ghrelin}} | |||
{{r|hypothalamus}} | |||
{{r|arcuate nucleus}} | |||
{{r|hunger}} | |||
{{r|satiety}} | |||
{{r|cholecystokinin}} | |||
==Articles related by keyphrases (Bot populated)== | |||
{{r|Gout}} | |||
{{r|Diabetes mellitus type 2}} | |||
{{r|Hypercholesterolemia}} | |||
Latest revision as of 11:01, 16 July 2024
- See also changes related to Bariatric surgery, or pages that link to Bariatric surgery or to this page or whose text contains "Bariatric surgery".
Related topics
- Circadian rhythms and appetite [r]: Daily variations in the regulation of food intake. [e]
- Diabesity [r]: A term referring to the intricate relationship between type 2 diabetes and obesity. [e]
- Drug treatments for obesity [r]: Treatments of obesity that are based on drugs. [e]
- Energy balance in pregnancy and lactation [r]: Adaptations in the control of food intake and energy expenditure in different reproductive states. [e]
- Evolution of appetite regulating systems [r]: Comparisons of the mechanisms regulating food intake and energy expenditure between species. [e]
- Exercise and body weight [r]: Correlation between physical activity and the body mass index. [e]
- Food reward [r]: The brain mechanisms involved in reinforcing feeding behaviour. [e]
- Genetics of obesity [r]: The evidence for a genetic component to obesity in humans. [e]
- Gut-brain signalling [r]: The interaction between the gastrointestinal tract and the brain. [e]
- Health consequences of obesity [r]: Long-term effects of obesity on health. [e]
- Glucostatic theory of appetite control [r]: The theory that changes in blood glucose concentrations or arteriovenous glucose differences are detected by glucoreceptors that affect energy intake. [e]
- Melanocortins and appetite [r]: The regulation of food intake through neuropeptides related to adrenocorticotropic hormone. [e]
- Stress and appetite [r]: The interactions between the hypothalamo-pituitary-adrenal axis and the regulation of food intake. [e]
- Adipocyte [r]: Cell that stores fat and makes it available for use as energy. [e]
- Leptin [r]: Hormone secreted by adipocytes that regulates appetite. [e]
- Ghrelin [r]: A hormone produced by P/D1 cells lining the fundus of the human stomach that stimulate appetite. [e]
- Hypothalamus [r]: A part of the mammalian brain located below the thalamus, forming the major portion of the ventral region of the diencephalon. [e]
- Arcuate nucleus [r]: An aggregation of neurons in the mediobasal hypothalamus with important roles in appetite regulation and in the control of growth hormone secretion and prolactin secretion. [e]
- Hunger [r]: Localized subjective sensation, caused by emptiness and a resulting hypermotility of the stomach. [e]
- Satiety [r]: Add brief definition or description
- Cholecystokinin [r]: Peptide hormone synthesised by L-cells in the mucosal epithelium of the duodenum, and secreted in response to the presence of partially digested lipids and proteins. [e]
- Gout [r]: Disturbance of uric-acid metabolism, characterized by painful inflammation of the joints, especially of the feet, from the deposition of urate crystals. [e]
- Diabetes mellitus type 2 [r]: Medical condition characterised by glucose intolerance and hyperglycemia [e]
- Hypercholesterolemia [r]: Presence of an abnormally large amount of cholesterol in the cells and plasma of the circulating blood. [e]